Antagonism is hostility that results in active resistance, opposition, or contentiousness. Wikipedia does not have an article with this exact name. ... Wiktionary (a portmanteau of wiki and dictionary) is a multilingual, Web-based project to create a free content dictionary, available in over 150 languages. ...
Additionally, it may refer to:
Antagonism (chemistry), where the involvement of multiple agents reduce their overall effect
Antagonism (philosophy), a principle, force or factor that is an active resistance, opposition, or contentiousness
Antagonism (pharmacology), when a substance binds to the same site an agonist would bind to without causing activation of the receptor
In chemistry, antagonism is a phenomenon where two or more agents in combination have an overall effect which is less than the sum of their individual effects. ... In phytopathology, antagonism refers to the suppression or interference with the normal growth of a plant pathogen by another microorganism, such as bacteria or fungi. ...
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The separation and temporary delay is occasioned by the weakening of the psycho-magnet of the spirit; in a strong spirit it develops into antagonism, in a weak one into submission.
Just as the third dimension does not nullify the laws of the plane, but is infinitely larger than the latter, so also the laws of spiritual knowledge encompass all your laws, being infinitely broader.
Antagonism to Truth infects space, but there is still more loathsome action when people, after having once realized Truth, later shrink from it.
Thus, RARs may antagonize AP1 function by binding (directly or indirectly) to c-Jun/c-Fos to form an inactive complex or may interact with and sequester another nuclear accessory factor that is required for AP1-mediated transcriptional activation(3).
Given the observed differences in the regions of the RARs associated with transcriptional activation and AP1 antagonism, it is possible that the structural features of the RARs required for the protein-DNA interactions leading to transcriptional activation may be somewhat different from those required for the protein-protein interactions involved in AP1 antagonism.
Antagonism of AP1 action also explains RA inhibition of collagenase production by fibroblasts, monocytes, and keratinocytes (29, 30, 31) and it may be the underlying mechanism for the therapeutic effect of RA on photodamaged human skin (32).
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