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BZP was originally synthesized in 1944 as a potential anthelmintic (anti-parasitic) agent.
BZP probably has amphetamine-like actions on the serotonin reuptake transporter, which increase serotonin concentrations in the extracellular fluids surrounding the cell and thereby increasing activation of the surrounding serotonin receptors.
BZP is an antagonist at the receptor, like yohimbine, which inhibits negative feedback, causing an increase in released noradrenaline.
BZP acts as a stimulant in humans and produces euphoria and cardiovascular effects, namely increases in heart rate and systolic blood pressure.
BZP is often taken in combination with 1-[3-(trifluoro-methyl)phenyl]piperazine (TFMPP), a noncontrolled substance, in order to enhance its spectrum of effects and has been promoted to youth population as substitute for MDMA at raves (all-night dance parties).
BZP is encountered as pink, white, off-white, purple, orange, tan, and mottle orange-brown tablets.
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